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Objective: In this study, we utilized bibliometric methods to assess the worldwide scientific output and identify hotspots related to the research on the lee vrac anion channel VRAC from to To analyzed the data, we utilized VOSviewer, lee vrac, a tool for visualizing network, to create networks based on the collaboration between countries, institutions, and authors. Additionally, we performed an analysis of journal co-citation, document citation, and co-occurrence of keywords.

Federal government websites often end in. The site is secure. The original contributions presented in the study are included in the article, further inquiries can be directed to the corresponding author. Objective: In this study, we utilized bibliometric methods to assess the worldwide scientific output and identify hotspots related to the research on the volume-regulated anion channel VRAC from to To analyzed the data, we utilized VOSviewer, a tool for visualizing network, to create networks based on the collaboration between countries, institutions, and authors.

Lee vrac

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This study offers insights lee vrac the investigation of VRAC channel and may guide researchers in identifying new directions for future research. Among these countries, the United States boasted the greatest quantity of publications and citations.

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Mogin et al. B Co-authorship network map of institutions with over two publications. Different colors indicated the variety of clusters. Schlichter, L. CysLT1 receptor antagonists pranlukast and zafirlukast inhibit LRRC8-mediated volume regulated anion channels independently of the receptor. During this period, various molecular candidates for VRAC were proposed. Figure 4B depicts the co-authorship network involving institutions with more than two papers to the VRAC research field. All literature retrieval and record downloads were collected in a single day 22 March to eliminate any potential deviation resulting from daily updates to the database. Two conventional protein kinase C isoforms, alpha and beta I, are involved in the ATP-induced activation of volume-regulated anion channel and glutamate release in cultured astrocytes. We utilized the WoS Literature Analysis Report online platform to conduct a comprehensive analysis of various publication characteristics. Cell 80 , — The co-occurrence analysis included keywords that appeared at least twice were included in three visualizations: network, overlay and density. VRAC has become a topic of increasing interest, and our analysis shows that it continues to be a prominent area. R6 Advanced to analyzed keywords and co-cited references with the strongest burst. Ion channels in cancer: Are cancer hallmarks oncochannelopathies?

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Qiu, Z. Disruption of ClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus. TABLE 3. Among the cited references, the article by Voss et al. For example, besides cancer and ischemic conditions after stroke, VRAC modulators were recently reported to be beneficial for murine type 2 diabetes Gunasekar et al. CLC chloride channels and transporters: Structure, function, physiology, and disease. Sabirov 8 It has been connected to different types of cancer including gastric cancer, glioblastoma, hepatocellular carcinoma and it has been implicated in anti-tumor drug resistance by its role in cellular drug uptake. Functional and pharmacological characterization of volume-regulated anion channels in human normal and cystic fibrosis bronchial and nasal epithelial cells. Among widely utilized bibliometric visualization tools, VOSviewer and CiteSpace have emerged as popular choices for data analysis. Ion channel dysfunction is increasingly associated with cancer Prevarskaya et al. Pharmacology of Ion Channels and Channelopathies. Green, J. It has been associated with various diseases including epilepsy, leukodystrophy, atherosclerosis, hypertension, diabetes, cerebral edema and stroke. Figure 7A illustrated that VRAC research analysis revealed 72 documents that received more than 30 citations.